hi every body

hi to all .
i know you aree not used to these greetings here. anyway how are you?
i care if you are well or not!
enjoy your life to the fullest because there is only one life that you get.
do good things in you life and with your life.
make yourself stand out in this billions of crowd.
make your life enjoyable and worth rememberiing.
bye for now . i will be back with more guff to share . in a hurry kinda situation.
thanks

22nd birth day

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Gene defect explains high blood pressure

bbc
High blood pressure could be explained by a genetic "fault" which means tiny blood vessels cannot relax, scientists say.US researchers said the finding could lead to a genetic test which could allow the development of new ways of diagnosing or treating the disease.People with high blood pressure, called hypertension, are at higher risk of having a stroke or heart attack, and can suffer other health problems.Blood pressure is the force exerted by the blood against artery walls."Developing a test for these abnormalities may lead to new means of diagnosing or treating the disease"Kendall Blumer, Washington University School of MedicineA range of medications are available, but often doctors cannot determine which one is most suitable for a patient because the underlying cause of hypertension is unknown in 90 to 95% of cases.Maintaining pressureScientists do know that certain hormones and neurotransmitters, which transmit nerve impulses, send out signals which cause tiny arteries known as arterioles to constrict, causing blood pressure to rise.They also help to regulate blood pressure by altering the levels of electrolyte, liquid that conducts electricity, and fluid in the kidneys.However scientists did not know how the hormones and neurotransmitters maintained normal blood pressure when conditions changed, such as when a person suddenly stood up or started running.It was thought a protein called "regulator of G protein signalling 2" (RGS2) was involved in blood pressure regulation.Researchers from Washington University School of Medicine looked at mice who lacked both copies of the RGS2 gene, which is responsible for RGS2 production.They also looked at mice which only lacked one copy of the gene.Both groups were compared to normal mice.It was found that all the mice who lacked one or both of the genes had very high blood pressure.The researchers say their finding that even those missing one gene were hypertensive shows the protein is crucial for controlling blood pressure.'Insights'Further tests showed it was possible to treat the mice with the genetic defect by giving them substances which block the action of a hormone called angiotensin II, which causes arterioles to constrict, increasing blood pressure.The researchers say this suggests RGS2 stops the action of angiotensin II, enabling arterioles to relax and blood pressure to decline.If there is little or no RGS2, the effect of angiotensin II is prolonged, so blood pressure remains high, they say.Kendall Blumer, professor of cell biology and physiology at Washington University School of Medicine, who led the research, said: "These findings provide new insights into the cause of hypertension and how normal blood pressure is regulated."This may lead to a way of determining the underlying cause of a person's hypertension and the most effective treatment for that individual."He added: "We hypothesise that genetic defects or differences that affect the amount or function of this protein may increase the risk of developing hypertension."Developing a test for these abnormalities may lead to new means of diagnosing or treating the disease."The team plan to carry out further research to see exactly how RGS2 works.The research is published in the Journal of Clinical Investigation.

Robot to carry out heart surgery

Friday, 20 July 2007, 10:16 GMT 11:16 UKbbc

The robotic arm is controlled from a computer consoleA robotic arm able to carry out an intricate life-saving heart operation is being pioneered by UK surgeons

The robot is used to guide thin wires through blood vessels in the heart to treat a fast or irregular heartbeat.Doctors at St Mary's Hospital in London say it will reduce risk for patients and increase the number of procedures they can carry out.More than 20 patients have been operated on with the robot, which is only one of four in use in the world.In the UK a shortage of expertise means there are too few centres where highly complex cases can be carried outDr Wyn DaviesDuring the procedure, known as catheter ablation, several thin wires and tubes are inserted through a vein in the groin and guided into the heart where they deliver an electric current to specific areas of heart muscle.The electric current destroys tiny portions of heart tissue which are causing the abnormal heartbeat.With the Sensei robot, surgeons use a joystick on a computer console to more accurately position and control the wires, which often need to be placed in locations that are difficult to reach.In the future the system could be automated so the robot guides the wires to a point in the heart selected by the doctor from images on a computer screen.Poor accessWhen done by hand the operation is highly skilled and a shortage of clinicians able to carry out the surgery means only 10% of people with the condition, called atrial fibrillation, are treated this way.Tony Blair underwent the operation by hand in 2004.Around 50,000 people develop atrial fibrillation, which is a major cause of strokes and heart failure, every year and it has been calculated to cost the NHS almost 1% of its entire annual budget.Numbers are expected to increase even more due to an ageing population, a rising number of people with chronic heart disease and better diagnosis.St Mary's consultant cardiologist, Dr Wyn Davies, said: "In the UK a shortage of expertise means there are too few centres where highly complex cases can be carried out."The robot allows accuracy and control of catheter movement which cannot currently be achieved without a skill level that usually takes considerable time to acquire.""The attraction is the potential for automation - we can get details about the patient's heart anatomy from CT scans, then on the computer draw where you want the ablation delivered and hit return."He added that full automation was a few years away but he could envisage a scenario where a skilled operator could oversee multiple operations happening at once.Trudie Lobban, chief executive of the Arrhythmia Alliance charity said the operation was highly successful and allowed people to lead normal lives."It's like threading cotton through a very fine needle and with this new device it should be much easier and quicker to carry out and to train doctors to do it."Professor Peter Weissberg, medical director of the British Heart Foundation, said: "Through research we have learned that abnormal heart rhythms, like atrial fibrillation, are caused by a handful of rogue cells."Extreme precision is required to track down and deal with these cells without damaging healthy tissue."The early promising results suggest that this approach may greatly improve the treatment of some patients with atrial fibrillation."

Hope arises for Down syndrome victims

HNSJAIPURJust one extra chromosome can blight a life, because it leads to Down syndrome, the commonest cause of mental retardation and malformation among newborns. There is no treatment. But now there may be. City paediatrician Sunil Gupta after 20 years’ research claims to have found a new way to treat Down syndrome victims, treat the “disturbed” cells in the human body and increase the proportion of “perfect” cells.Gupta combines modern medicine with traditional Indian methods in his treatment. So far he has treated over 300 Down syndrome patients in the age group 1 to 28, from all parts of India.According to Gupta, there are three types of cells in the human body - perfect cells, damaged cells and cells in a disturbed condition. “I just work on those disturbed cells.I try to get them to work properly and increase the number of perfect cells in the body,” he says. Down syndrome occurs in about one out of every 800 to 1,000 births. It affects boys and girls equally. It is caused by the presence of 47 chromosomes in the newborn, instead of the usual 46.Almost all parents who hear their newborn child has Down syndrome are devastated. Take the case of the Mathurs (name changed). For 15 years the Mathurs had to see their only son live like a vegetable, completely dependent on them. But the condition of the boy has improved considerably after treatment by Gupta. He has even passed an examination for tabla players from a school of classical music.The parents have new plans for their boy. “We are looking for an instructor who can help him establish some business,” says the father. His mother says, “I’ve started dreaming about my daughter-in-law. As he is doing well and he is caring too, I’ll get him married as soon he turns an adult.”Gupta says, “The number of mentally retarded children in India has crossed the five million mark. Their rehabilitation is going to remain a challenge unless the government and voluntary agencies make concerted efforts. These children need treatment and they also need help so that they can become self-reliant.” The paediatrician faced a problem when he tried to patent the medicine he uses. “I wanted to get my herbal formula patented in India, but the patent unit of CSIR (Council for Scientific and Industrial Research) said it could not be done in India. But now an American pharmaceutical company will market Gupta’s composition.“I have signed an agreement with an American company called Nutrivene, which will market a nutritional supplement composed on the basis of my herbal formula for retarded children and Down syndrome victims,” he said.

Special pills to control hunger

HNSLONDON:Scientists in Italy claim to have developed a pill that controls hunger for a few hours and may help overweight people in their battle against the bulge.The tiny pill, which is yet to be named, is made from a hydrogel. It is powdery when dry but turns to a jelly-like ball in the stomach when swallowed, reported the online edition of BBC News.Made from an organic compound called cellulose, it is easily flushed out by the body and can stop hunger for a few hours. It makes dieters feel full, said scientists at Italy’s National Research Council. As part of the study, one of the researchers tried the pill at about 11 a.m. and was still feeling full at six in the evening, they said. The pill should be taken about 30 minutes to one hour before each meal. People could still eat a meal but would need only small portions. The pill passes through the digestive system within five to six hours. The researchers have already tested it on 20 people. It is also being tested on 90 other overweight volunteers who will be monitored to see how much weight they lose and if there are any adverse effects. The results are expected in October this year.The scientists expect the product to be launched in May 2008

Japanese clone frozen mice

Tokyo (dbTechno) - Scientists in Japan have successfully cloned dead mice that were frozen for a period of up to 16 years.The dead mice have been frozen for nearly two decades, but geneticists were able to successfully clone them.Researchers from the Center for Developmental Biology of Japan’s RIKEN research institute carried out the work.The team was led by Teruhiko Wakayama and used a technique that involved nuclear transfer.Using this, they were able to basically remove the nucleus of an egg cell, and then replace it with a cell from the frozen ice.They were then able to spark a reaction that made the cell begin to divide, thus creating the clone.The research has been published in the Proceedings of the National Academy of Sciences.Scientists in Japan clone mice dead for 16 yearsNovember 4, 2008 ·
We raised the issue of biodiversity loss earlier today. Basically, the concern is that species extinctions are not just a warning flag, but also pose an inherent problem, in of itself. Although it is not really being proposed as a solution to this problem, scientists in Japan have recently announced the cloning of mice that had been dead for 16 years.While animals have already been cloned by scientists, this new process is different since it takes animals that have been dead and frozen for a number of years. Normally, when a cell is frozen it bursts, which has up until now made it impossible to clone frozen animals (like wooly mammoths). With this new process, there is the possibility to clone long-dead creatures that have been frozen.In theory, this could allow scientists to freeze animals that are on the verge of extinction to be cloned in the future when environmental conditions improve. However, this is pure speculation on our part, and would, of course, not address the more immediate problems stemming from loss of biodiversity.